p prkcb Search Results


p prkcd  (Cell Signaling Technology Inc)
95
Cell Signaling Technology Inc p prkcd
P Prkcd, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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prkcd p s663  (Cell Signaling Technology Inc)
94
Cell Signaling Technology Inc prkcd p s663
Prkcd P S663, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti-phosphorylated pkcbii 2252-1  (Danaher Inc)
90
Danaher Inc anti-phosphorylated pkcbii 2252-1
Anti Phosphorylated Pkcbii 2252 1, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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4292 pkcalpha cell signaling 2056 pkd pkcmu cell signaling 2052 p pkcalpha betaii  (Cell Signaling Technology Inc)
98
Cell Signaling Technology Inc 4292 pkcalpha cell signaling 2056 pkd pkcmu cell signaling 2052 p pkcalpha betaii
4292 Pkcalpha Cell Signaling 2056 Pkd Pkcmu Cell Signaling 2052 P Pkcalpha Betaii, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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4292 pkcalpha cell signaling 2056 pkd pkcmu cell signaling 2052 p pkcalpha betaii - by Bioz Stars, 2026-03
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p prkcb s660  (Cell Signaling Technology Inc)
95
Cell Signaling Technology Inc p prkcb s660
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P Prkcb S660, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/p prkcb s660/product/Cell Signaling Technology Inc
Average 95 stars, based on 1 article reviews
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p prkcq t538  (Cell Signaling Technology Inc)
93
Cell Signaling Technology Inc p prkcq t538
iHPCAL1 protects against ferroptosis-associated acute pancreatitis in mice. (a) Representative images of pancreatic histology in cerulein-induced pancreatitis in Gpx4 wild-type (WT) and KO mice with or without iHPCAL1 treatment (10 mg/kg; bar = 200 µm). Histological scores for acinar cell death, leukocyte infiltration, and edema at 12 h after the last cerulein treatment were evaluated. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (b-m) In parallel, serum AMY (b), pancreatic trypsin activity (c), pancreatic MPO activity (d), serum LDH (e), serum HMGB1 (f), serum TNF (g), serum IL6 (h), serum IL1B (I), pancreatic Ptgs2 mRNA (j), pancreatic MDA (k), pancreatic CASP3 activity (l), and pancreatic HPCAL1 protein (m) were assayed at 12 h after the last cerulein treatment. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (n) Schematic depicting the role of HPCAL1 in the promotion of ferroptosis by mediating the autophagic degradation of CDH2. Ferroptosis activators (e.g., erastin and RSL3), but not apoptosis inducers (e.g., STS) or classic autophagy inducers (e.g., HBSS and rapamycin) induce PRKCQ phosphorylation on <t>T538,</t> which leads to HPCAL1 phosphorylation on T149. Phosphorylated HPCAL1 acts as a selective autophagy receptor for CDH2 and mediates the degradation of CDH2 in lysosomes. The loss of CDH2 decreases mechanotransduction and ultimately reduces cell connections, thereby accelerating the lipid peroxidation of ferroptosis.
P Prkcq T538, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
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anti-p-prkca antibody  (Affinity Biosciences)
90
Affinity Biosciences anti-p-prkca antibody
iHPCAL1 protects against ferroptosis-associated acute pancreatitis in mice. (a) Representative images of pancreatic histology in cerulein-induced pancreatitis in Gpx4 wild-type (WT) and KO mice with or without iHPCAL1 treatment (10 mg/kg; bar = 200 µm). Histological scores for acinar cell death, leukocyte infiltration, and edema at 12 h after the last cerulein treatment were evaluated. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (b-m) In parallel, serum AMY (b), pancreatic trypsin activity (c), pancreatic MPO activity (d), serum LDH (e), serum HMGB1 (f), serum TNF (g), serum IL6 (h), serum IL1B (I), pancreatic Ptgs2 mRNA (j), pancreatic MDA (k), pancreatic CASP3 activity (l), and pancreatic HPCAL1 protein (m) were assayed at 12 h after the last cerulein treatment. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (n) Schematic depicting the role of HPCAL1 in the promotion of ferroptosis by mediating the autophagic degradation of CDH2. Ferroptosis activators (e.g., erastin and RSL3), but not apoptosis inducers (e.g., STS) or classic autophagy inducers (e.g., HBSS and rapamycin) induce PRKCQ phosphorylation on <t>T538,</t> which leads to HPCAL1 phosphorylation on T149. Phosphorylated HPCAL1 acts as a selective autophagy receptor for CDH2 and mediates the degradation of CDH2 in lysosomes. The loss of CDH2 decreases mechanotransduction and ultimately reduces cell connections, thereby accelerating the lipid peroxidation of ferroptosis.
Anti P Prkca Antibody, supplied by Affinity Biosciences, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti-p-prkca antibody/product/Affinity Biosciences
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and p-prkce (gtx105452, 1:500)  (GeneTex)
90
GeneTex and p-prkce (gtx105452, 1:500)
iHPCAL1 protects against ferroptosis-associated acute pancreatitis in mice. (a) Representative images of pancreatic histology in cerulein-induced pancreatitis in Gpx4 wild-type (WT) and KO mice with or without iHPCAL1 treatment (10 mg/kg; bar = 200 µm). Histological scores for acinar cell death, leukocyte infiltration, and edema at 12 h after the last cerulein treatment were evaluated. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (b-m) In parallel, serum AMY (b), pancreatic trypsin activity (c), pancreatic MPO activity (d), serum LDH (e), serum HMGB1 (f), serum TNF (g), serum IL6 (h), serum IL1B (I), pancreatic Ptgs2 mRNA (j), pancreatic MDA (k), pancreatic CASP3 activity (l), and pancreatic HPCAL1 protein (m) were assayed at 12 h after the last cerulein treatment. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (n) Schematic depicting the role of HPCAL1 in the promotion of ferroptosis by mediating the autophagic degradation of CDH2. Ferroptosis activators (e.g., erastin and RSL3), but not apoptosis inducers (e.g., STS) or classic autophagy inducers (e.g., HBSS and rapamycin) induce PRKCQ phosphorylation on <t>T538,</t> which leads to HPCAL1 phosphorylation on T149. Phosphorylated HPCAL1 acts as a selective autophagy receptor for CDH2 and mediates the degradation of CDH2 in lysosomes. The loss of CDH2 decreases mechanotransduction and ultimately reduces cell connections, thereby accelerating the lipid peroxidation of ferroptosis.
And P Prkce (Gtx105452, 1:500), supplied by GeneTex, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/and p-prkce (gtx105452, 1:500)/product/GeneTex
Average 90 stars, based on 1 article reviews
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circ-prkci  (CH Instruments)
90
CH Instruments circ-prkci
iHPCAL1 protects against ferroptosis-associated acute pancreatitis in mice. (a) Representative images of pancreatic histology in cerulein-induced pancreatitis in Gpx4 wild-type (WT) and KO mice with or without iHPCAL1 treatment (10 mg/kg; bar = 200 µm). Histological scores for acinar cell death, leukocyte infiltration, and edema at 12 h after the last cerulein treatment were evaluated. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (b-m) In parallel, serum AMY (b), pancreatic trypsin activity (c), pancreatic MPO activity (d), serum LDH (e), serum HMGB1 (f), serum TNF (g), serum IL6 (h), serum IL1B (I), pancreatic Ptgs2 mRNA (j), pancreatic MDA (k), pancreatic CASP3 activity (l), and pancreatic HPCAL1 protein (m) were assayed at 12 h after the last cerulein treatment. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (n) Schematic depicting the role of HPCAL1 in the promotion of ferroptosis by mediating the autophagic degradation of CDH2. Ferroptosis activators (e.g., erastin and RSL3), but not apoptosis inducers (e.g., STS) or classic autophagy inducers (e.g., HBSS and rapamycin) induce PRKCQ phosphorylation on <t>T538,</t> which leads to HPCAL1 phosphorylation on T149. Phosphorylated HPCAL1 acts as a selective autophagy receptor for CDH2 and mediates the degradation of CDH2 in lysosomes. The loss of CDH2 decreases mechanotransduction and ultimately reduces cell connections, thereby accelerating the lipid peroxidation of ferroptosis.
Circ Prkci, supplied by CH Instruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/circ-prkci/product/CH Instruments
Average 90 stars, based on 1 article reviews
circ-prkci - by Bioz Stars, 2026-03
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p pkc  (Cell Signaling Technology Inc)
94
Cell Signaling Technology Inc p pkc
iHPCAL1 protects against ferroptosis-associated acute pancreatitis in mice. (a) Representative images of pancreatic histology in cerulein-induced pancreatitis in Gpx4 wild-type (WT) and KO mice with or without iHPCAL1 treatment (10 mg/kg; bar = 200 µm). Histological scores for acinar cell death, leukocyte infiltration, and edema at 12 h after the last cerulein treatment were evaluated. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (b-m) In parallel, serum AMY (b), pancreatic trypsin activity (c), pancreatic MPO activity (d), serum LDH (e), serum HMGB1 (f), serum TNF (g), serum IL6 (h), serum IL1B (I), pancreatic Ptgs2 mRNA (j), pancreatic MDA (k), pancreatic CASP3 activity (l), and pancreatic HPCAL1 protein (m) were assayed at 12 h after the last cerulein treatment. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (n) Schematic depicting the role of HPCAL1 in the promotion of ferroptosis by mediating the autophagic degradation of CDH2. Ferroptosis activators (e.g., erastin and RSL3), but not apoptosis inducers (e.g., STS) or classic autophagy inducers (e.g., HBSS and rapamycin) induce PRKCQ phosphorylation on <t>T538,</t> which leads to HPCAL1 phosphorylation on T149. Phosphorylated HPCAL1 acts as a selective autophagy receptor for CDH2 and mediates the degradation of CDH2 in lysosomes. The loss of CDH2 decreases mechanotransduction and ultimately reduces cell connections, thereby accelerating the lipid peroxidation of ferroptosis.
P Pkc, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/p pkc/product/Cell Signaling Technology Inc
Average 94 stars, based on 1 article reviews
p pkc - by Bioz Stars, 2026-03
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p prkcz i  (Cell Signaling Technology Inc)
94
Cell Signaling Technology Inc p prkcz i
Reagent sources.
P Prkcz I, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
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p prkca b  (Cell Signaling Technology Inc)
95
Cell Signaling Technology Inc p prkca b
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P Prkca B, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 95 stars, based on 1 article reviews
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Image Search Results


Reagent sources.

Journal: Autophagy

Article Title: Identification of HPCAL1 as a specific autophagy receptor involved in ferroptosis

doi: 10.1080/15548627.2022.2059170

Figure Lengend Snippet: Reagent sources.

Article Snippet: p-PRKCB (S660) , Cell Signaling Technology , 9371 RRID: AB_2168219.

Techniques: Recombinant, Lysis, Protease Inhibitor, BIA-KA, Cell Fractionation, ROS Assay, Enzyme-linked Immunosorbent Assay, Iron Assay, cDNA Synthesis, Lactate Dehydrogenase Assay, Multiple Displacement Amplification, Membrane, Mutagenesis, Activity Assay, shRNA, Software

iHPCAL1 protects against ferroptosis-associated acute pancreatitis in mice. (a) Representative images of pancreatic histology in cerulein-induced pancreatitis in Gpx4 wild-type (WT) and KO mice with or without iHPCAL1 treatment (10 mg/kg; bar = 200 µm). Histological scores for acinar cell death, leukocyte infiltration, and edema at 12 h after the last cerulein treatment were evaluated. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (b-m) In parallel, serum AMY (b), pancreatic trypsin activity (c), pancreatic MPO activity (d), serum LDH (e), serum HMGB1 (f), serum TNF (g), serum IL6 (h), serum IL1B (I), pancreatic Ptgs2 mRNA (j), pancreatic MDA (k), pancreatic CASP3 activity (l), and pancreatic HPCAL1 protein (m) were assayed at 12 h after the last cerulein treatment. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (n) Schematic depicting the role of HPCAL1 in the promotion of ferroptosis by mediating the autophagic degradation of CDH2. Ferroptosis activators (e.g., erastin and RSL3), but not apoptosis inducers (e.g., STS) or classic autophagy inducers (e.g., HBSS and rapamycin) induce PRKCQ phosphorylation on T538, which leads to HPCAL1 phosphorylation on T149. Phosphorylated HPCAL1 acts as a selective autophagy receptor for CDH2 and mediates the degradation of CDH2 in lysosomes. The loss of CDH2 decreases mechanotransduction and ultimately reduces cell connections, thereby accelerating the lipid peroxidation of ferroptosis.

Journal: Autophagy

Article Title: Identification of HPCAL1 as a specific autophagy receptor involved in ferroptosis

doi: 10.1080/15548627.2022.2059170

Figure Lengend Snippet: iHPCAL1 protects against ferroptosis-associated acute pancreatitis in mice. (a) Representative images of pancreatic histology in cerulein-induced pancreatitis in Gpx4 wild-type (WT) and KO mice with or without iHPCAL1 treatment (10 mg/kg; bar = 200 µm). Histological scores for acinar cell death, leukocyte infiltration, and edema at 12 h after the last cerulein treatment were evaluated. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (b-m) In parallel, serum AMY (b), pancreatic trypsin activity (c), pancreatic MPO activity (d), serum LDH (e), serum HMGB1 (f), serum TNF (g), serum IL6 (h), serum IL1B (I), pancreatic Ptgs2 mRNA (j), pancreatic MDA (k), pancreatic CASP3 activity (l), and pancreatic HPCAL1 protein (m) were assayed at 12 h after the last cerulein treatment. Data are presented as mean ± SD; n = 6 mice/group; one-way ANOVA test on all pairwise combinations. (n) Schematic depicting the role of HPCAL1 in the promotion of ferroptosis by mediating the autophagic degradation of CDH2. Ferroptosis activators (e.g., erastin and RSL3), but not apoptosis inducers (e.g., STS) or classic autophagy inducers (e.g., HBSS and rapamycin) induce PRKCQ phosphorylation on T538, which leads to HPCAL1 phosphorylation on T149. Phosphorylated HPCAL1 acts as a selective autophagy receptor for CDH2 and mediates the degradation of CDH2 in lysosomes. The loss of CDH2 decreases mechanotransduction and ultimately reduces cell connections, thereby accelerating the lipid peroxidation of ferroptosis.

Article Snippet: p-PRKCQ (T538) , Cell Signaling Technology , 9377 RRID: AB_2172071.

Techniques: Activity Assay, Phospho-proteomics

Reagent sources.

Journal: Autophagy

Article Title: Identification of HPCAL1 as a specific autophagy receptor involved in ferroptosis

doi: 10.1080/15548627.2022.2059170

Figure Lengend Snippet: Reagent sources.

Article Snippet: p-PRKCQ (T538) , Cell Signaling Technology , 9377 RRID: AB_2172071.

Techniques: Recombinant, Lysis, Protease Inhibitor, BIA-KA, Cell Fractionation, ROS Assay, Enzyme-linked Immunosorbent Assay, Iron Assay, cDNA Synthesis, Lactate Dehydrogenase Assay, Multiple Displacement Amplification, Membrane, Mutagenesis, Activity Assay, shRNA, Software

Reagent sources.

Journal: Autophagy

Article Title: Identification of HPCAL1 as a specific autophagy receptor involved in ferroptosis

doi: 10.1080/15548627.2022.2059170

Figure Lengend Snippet: Reagent sources.

Article Snippet: p-PRKCZ/I (T410/403) , Cell Signaling Technology , 9378 RRID: AB_2168217.

Techniques: Recombinant, Lysis, Protease Inhibitor, BIA-KA, Cell Fractionation, ROS Assay, Enzyme-linked Immunosorbent Assay, Iron Assay, cDNA Synthesis, Lactate Dehydrogenase Assay, Multiple Displacement Amplification, Membrane, Mutagenesis, Activity Assay, shRNA, Software

Reagent sources.

Journal: Autophagy

Article Title: Identification of HPCAL1 as a specific autophagy receptor involved in ferroptosis

doi: 10.1080/15548627.2022.2059170

Figure Lengend Snippet: Reagent sources.

Article Snippet: p-PRKCA/B (T638/641) , Cell Signaling Technology , 9375 RRID: AB_2284224.

Techniques: Recombinant, Lysis, Protease Inhibitor, BIA-KA, Cell Fractionation, ROS Assay, Enzyme-linked Immunosorbent Assay, Iron Assay, cDNA Synthesis, Lactate Dehydrogenase Assay, Multiple Displacement Amplification, Membrane, Mutagenesis, Activity Assay, shRNA, Software